The overarching goal of my research program is to define the signals that initiate and sustain aberrant cell
development leading to neuroendocrine cell plasticity. My ongoing research combines my expertise in transgenic
mouse models, 3-D organoid culture systems, and state-of-the-art sequencing technologies to elucidate the
underlying etiology of gastroenteropancreatic neuroendocrine neoplasms. I am highly motivated to translate
these discoveries toward defining personalized biomarkers and therapeutic approaches for these diseases with
limited options for screening and treatment.